Download Free Medical Mitra Valvular Disease PowerPoint Presentation

Mitra Valvular Disease PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical PowerPoint presentation titled "MITRA VALVULAR DISEASE" by Dr. Swarnalatha Devi MD.

This medical PowerPoint presentation talks about Mitra valvular disease. Mitra valvular disease, also known as mitral valve disease or mitral valve regurgitation, is a condition in which the mitral valve of the heart does not close properly, leading to the backflow of blood into the left atrium. This can result in a range of symptoms including fatigue, shortness of breath, chest pain, palpitations, and heart failure.

Tags : Mitra valvular disease, mitral valve disease, mitral valve regurgitation, shortness of breath, chest pain, palpitations, heart failure, cardiology

Published on : Mar 17, 2023 | Views : 933 | Downloads : 1

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

MITRA VALVULAR DISEASE
Slide 2-

Dr. Swarnalatha Devi MD Professor of Medicine
Slide 3-

MITRAL STENOSIS Aetiology and pathophysiology Mitral stenosis is almost always rheumatic in origin, although in the elderly it can be caused by heavy calcification of the mitral valve apparatus. There is also a rare from of congenital mitral stenosis.
Slide 4-

AETIOLOGY Rheumatic heart disease. Congenital—parachute mitral valve. Hunter”, hurler” syndrome. Drugs---methysergide. Carcinoid syndrome.
Slide 5-

PATHO PHYSIOLOGY In rheumatic mitral stenosis, the mitral valve orifice is slowly diminished by progressive fibrosis, calcification of the valve leaflets, and fusion of the cusps and subvalvular apparatus. The flow of blood from left atrium to left ventricle is restricted and left atrial pressure rises, leading to pulmonary venous congestion and breathlessness. There is dilatation and hypertrophy of the left atrium, and left ventricular filling becomes more dependent on left atrial contraction.
Slide 6-

PATHOPHYSIOLOGY Any increase in heart rate shortens diastole when the mitral valve has to open it produces further rise in left atrial pressure; Situations that demand an increase in cardiac output will also increase left atrial pressure. Exercise and pregnancy are therefore poorly tolerated.
Slide 7-

The mitral valve orifice is 4 -5 cm2 in diastole and may be reduced to 1 cm2 or less in severe mitral stenosis. Patients usually remain asymptomatic until the stenosis is approximately 2 cm2 or less. At first, symptoms may occur only on exercise, in severe stenosis, left atrial pressure is permanently elevated so breathlessness occurs even at rest due to.Chronic pulmonary venous congestion. Low cardiac output may cause fatigue.
Slide 8-

Pulmonary hypertension may lead to right ventricular hypertrophy and dilatation, tricuspid regurgitation and right heart failure. Less than 20% of patients remain in sinus rhythm; many of these have a small fibrotic left atrium and severe pulmonary hypertension. All patients with mitral stenosis, and particularly those with atrial fibrillation, are at risk from left atrial thrombosis and systemic thromboembolism. .
Slide 9-

SYMPTOMS Dyspnoea. Palpitations. Fatigue. Hemoptysis. Recurrent bronchitis.
Slide 10-

CLINICAL EXAMINATION MITRAL FACIES. SMALL VOLUME PULSE. LEFT PARASTERNAL HEAVE. TAPPING APICAL IMPULSE (PALPABLE S1)
Slide 11-

Loud S1 ,opening snap,(in pliable valve). This is due to diastolic buckling of anterior mitral or tricuspid leaflet due to elevated left or right atrial pressure. A low pitched ,rough ,rumbling mid diastolic murmur with pre systolic accentuation best heard over apex, with bell of stethoscope in left lateral position,in expiration. ESM over pulmonary area if there is PAH. Pan systolic murmur in tricuspid area in functional TR.
Slide 12-

© Continuing Medical Implementation …...bridging the care gap Mitral Stenosis: Physical Exam First heart sound (S1) is accentuated and snapping Opening snap (OS) after aortic valve closure Low pitch diastolic rumble at the apex Pre-systolic accentuation (esp. if in sinus rhythm) S1 S2 OS S1
Slide 13-

INVESTIGATIONS ECG—”P” MITRALE AND RIGHT VENTRICULAR HYPERTROPHY. ATIAL FIBRILLATION ---ABSENT P WAVES ,VARYING R-R INTERVALS. X—RAY CHEST—LEFT ATRIAL ENLARGEMENT STRAIGHTENING OF LEFT BORDER KERLY A, B LINES R A O WITH BARIUM SWALLOW-COMPRESSION BY LEFT ATRIUM
Slide 14-

© Continuing Medical Implementation …...bridging the care gap A 75 year old woman with loud first heart sound and mid-diastolic murmer
Slide 15-

ECHO CARDIOGRAM—THICKENED ,IMMOBILE CUSPS, REDUCED RATE OF DIASTOLIC FILLING. REDUCED VALVE AREA. CARDIAC CATHETERISATION— PRESSURE GRADIENT BETWEEN LA AND LV
Slide 16-

ppt slide no 16 content not found
Slide 17-

Mitral Stenosis
Slide 18-

SEVERITY ACCORDING TO VALVE AREA---- -4-6 cm2-----------NORMAL > 2.5 cm2-----------NO SYMPTOMS 1.5—2.5 cm2---MILD -DYSPNOEA ON EXERTION 1-1.5 cm2---MODERATE- PND +P .EDEMA <1cm2---SEVERE /CRITICAL -ORTHOPNEA –CLASS 1V
Slide 19-

ACCORDING TO A2—OS INTERVAL IN SEVERE MS---- THIS PERIOD IS SHORTENED.—0.05-0.07 SEC MILD MS— 0.10-0.12 SEC ACCORDING TO PRESSURE GRADIENT ACROSS MITRAL VALVE— MILD-<5 mm Hg MODERATE—5—15 mm Hg SEVERE-->15 mm Hg DURATION OF DIASTOLIC MURMUR IS DIRECTLY PROPORTIONAL TO THE SEVERITY
Slide 20-

COMPLICATIONS HEMOPTYSIS--- Due to pulmonary apoplexy. Acute LVF—pulmonary edema. Pulmonary congestion. Recurrent bronchitis. Pulmonary infarction.
Slide 21-

ATRIAL FIBRILLATION-- 2/3 will have AF Pulse is irregularly irregular No a waves in JVP Intensity of S1 is variable AF precipitates---CCF, thrombo embolic phenomenon
Slide 22-

Infective endocarditis. Pressure symptoms----ortners syndrome----compression of recurrent laryngeal nerve by left atrium. Left bronchus--bronchiectasis.
Slide 23-

DIFFERENTIAL DIAGNOSIS Left atrial myxoma. Cortriatritum. Ball valve thrombus of left atrium. Flow murmurs of VSD, ASD, PDA. Carey-coombs murmur.
Slide 24-

Medical Management Anti failure measures. Anticoagulation in atrial fibrillation. Rheumatic fever prophylaxis. Infective endocarditis prophylaxis.
Slide 25-

SURGICAL TREATMENT Closed mitral valvotomy/commissurotomy—(with out cardio pulmonary by pass). Open mitral valvotomy/commissurotomy (with aid of cardio pulmonary by pass). Per cutaneous balloon valvuloplasty. Mitral valve replacement.
Slide 26-

Closed mitral valvotomy—in pliable valve and ,no mitral regurtitation Open mitral valvotomy---in this valve, sub valvular. C hordae tendinae are loosened, calcific deposits removed Thrombi if present are removed.
Slide 27-

Percutaneous balloon valvuloplasty---in pregnant women, Severe extracardiac disease. Criteria- -Significant symptoms Isolated MS. No mitral regurgitation. Mobile, non calcified valve. Normal sub valvular apparatus No thrombus in left atrium
Slide 28-

VALVE REPLACEMENT---- Associated MITRAL REGURGITATION Valve rigid, and calcified. Mechanical prosthesis— Caged ball valve(starr-edward prosthesis). TILTING DISK valves(st jude.Bjork-shiely valves)
Slide 29-

THANK YOU