Download Free Medical FLUID Therapy PowerPoint Presentation

FLUID Therapy PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical PowerPoint presentation titled "FLUID Therapy".

This medical PowerPoint presentation talks about Fluid therapy, a medical treatment that involves the administration of fluids to a patient in order to maintain or restore their fluid balance. It is commonly used in a variety of medical situations, including dehydration, shock, and surgical procedures.

The type and amount of fluids administered depend on the individual patients needs and medical condition. Common fluids used in fluid therapy include isotonic solutions, such as normal saline and lactated Ringers solution, as well as hypertonic and hypotonic solutions.

Fluid therapy can be given orally or intravenously, depending on the patients ability to tolerate fluids by mouth. It is usually monitored closely by healthcare professionals to ensure that the patient is receiving the appropriate amount and type of fluids.

In addition to restoring fluid balance, fluid therapy can also be used to deliver medications and other substances directly into the bloodstream, making it an important aspect of many medical treatments.

Tags : Fluid therapy, fluid balance, isotonic solutions, Emergency Medicine, Emergency Medicine PowerPOint presentation

Published on : Apr 03, 2023 | Views : 1569 | Downloads : 9

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

FLUID Therapy
Slide 2-

Fluid and electrolyte balance is an extremely complicated thing.
Slide 3-

Importance Need to make a decision regarding fluids in pretty much every hospitalized patient. Can be life-saving in certain conditions loss of body water, whether acute or chronic, can cause a range of problems from mild lightheadedness to convulsions, coma, and in some cases, death. Though fluid therapy can be a lifesaver, it's never innocuous, and can be very harmful.
Slide 4-

Kinds of IV Fluid solutions Hypotonic - 1/2NS Isotonic - NS, LR, albumen Hypertonic – Hypertonic saline. Crystalloid Colloid
Slide 5-

Crystalloid vs ColloidType of particles (large or small) Fluids with small “crystalizable” particles like NaCl are called crystalloids Fluids with large particles like albumin are called colloids, these don’t (quickly) fit through vascular pores, so they stay in the circulation and much smaller amounts can be used for same volume expansion. (250ml Albumin = 4 L NS) Edema resulting from these also tends to stick around longer for same reason. Albumin can also trigger anaphylaxis.
Slide 6-

There are two components to fluid therapy: Maintenance therapy replaces normal ongoing losses, and Replacement therapy corrects any existing water and electrolyte deficits.
Slide 7-

Maintenance therapy Maintenance therapy is usually undertaken when the individual is not expected to eat or drink normally for a longer time (eg, perioperatively or on a ventilator). Big picture: Most people are “NPO” for 12 hours each day. Patients who won’t eat for one to two weeks should be considered for parenteral or enteralnutrition.
Slide 8-

Maintenance Requirements can be broken into water and electrolyte requirements:
Slide 9-

Water — Two liters of water per day are generally sufficient for adults; Most of this minimum intake is usually derived from the water contentof food and the water of oxidation, therefore it has been estimated that only 500ml of water needs be imbibed given normal diet and no increased losses. These sources of water are markedly reduced in patients who are not eating and so must be replaced by maintenance fluids.
Slide 10-

water requirements increase with: fever, sweating, burns, tachypnea, surgical drains, polyuria, or ongoing significant gastrointestinal losses. For example, water requirements increase by 100 to 150 mL/day for each C degree of body temperature elevation.
Slide 11-

Several formulas can be used to calculate maintenance fluid rates.
Slide 12-

A comparison of formulas produces a widevariety of fluid recommendations: 2000 cc to 3378 cc for an obese woman who is65 inches tall and weighs 248 pounds (112.6 kg) This is a reminder that fluid needs, no matter what formula is used, are at best an estimation.
Slide 13-

4/2/1 rule a.k.a Weight+40 I prefer the 4/2/1 rule (with a 120 mL/h limit) because it is the same as for pediatrics.
Slide 14-

4/2/1 rule4 ml/kg/hr for first 10 kg (=40ml/hr)then 2 ml/kg/hr for next 10 kg (=20ml/hr)then 1 ml/kg/hr for any kgs over thatThis always gives 60ml/hr for first 20 kgthen you add 1 ml/kg/hr for each kg over 20 kg This boils down to: Weight in kg + 40 = Maintenance IV rate/hour.For any person weighing more than 20kg
Slide 15-

Maintenance IV rate:4/2/1 rule -> Weight in kg + 40
Slide 16-

What to put in the fluids
Slide 17-

Start: D5 1/2NS+20 meq K @ Wt+40/hr a reasonable approach is to start 1/2 normal saline to which 20 meq of potassium chloride is added per liter. (1/2NS+20 K @ Wt+40/hr) Glucose in the form of dextrose (D5) can be added to provide some calories while the patient is NPO. The normal kidney can maintain sodium and potassium balance over a wide range of intakes. So,start: D5 1/2NS+20 meq K at a rate equal to their weight + 40ml/hr, but no greater than 120ml/hr. then adjust as needed, see next page.
Slide 18-

Start D5 1/2NS+20 meq K, then adjust: If sodium falls, increase the concentration (eg, to NS) If sodium rises, decrease the concentration (eg, 1/4NS) If the plasma potassium starts to fall, add more potassium. If things are good, leave things alone.
Slide 19-

Usually kidneys regulate well, but:Altered homeostasis in the hospital In the hospital, stress, pain, surgery can alter the normal mechanisms. Increased aldosterone, Increased ADH They generally make patients retain more water and salt, increase tendency for edema, and become hypokalemic.
Slide 20-

ppt slide no 20 content not found
Slide 21-

ppt slide no 21 content not found
Slide 22-

ppt slide no 22 content not found
Slide 23-

ppt slide no 23 content not found
Slide 24-

Now onto Part 2 of the presentation:
Slide 25-

Hypovolemia Hypovolemia or FVD is result of water & electrolyte loss Compensatory mechanisms include: Increased sympathetic nervous system stimulation with an increase in heart rate & cardiac contraction; thirst; plus release of ADH & aldosterone Severe case may result in hypovolemic shock or prolonged case may cause renal failure
Slide 26-

Causes of FVD=hypovolemia: Gastrointestinal losses: N/V/D Renal losses: diuretics Skin or respiratory losses: burns Third-spacing: intestinal obstruction, pancreatitis
Slide 27-

Replacement therapy.
Slide 28-

A variety of disorders lead to fluid losses that deplete the extracellular fluid . This can lead to a potentially fatal decrease in tissue perfusion. Fortunately, early diagnosis and treatment can restore normovolemia in almost all cases.
Slide 29-

There is no easy formula for assessing the degree of hypovolemia. Hypovolemic Shock, the most severe form of hypolemia, is characterized by tachycardia, cold, clammy extremities, cyanosis, a low urine output (usually less than 15 mL/h), and agitation and confusion due to reduced cerebral blood flow. This needs rapid treatment with isotonic fluid boluses (1-2L NS), and assessment and treatment of the underlying cause. But hypovolemia that is less severe and therefore well compensated is more difficult to accurately assess.
Slide 30-

History for assessing hypovolemia The history can help to determine the presence and etiology of volume depletion. Weight loss! Early complaints include lassitude, easy fatiguability, thirst, muscle cramps, and postural dizziness. More severe fluid loss can lead to abdominal pain, chest pain, or lethargy and confusion due to ischemia of the mesenteric, coronary, or cerebral vascular beds, respectively. Nausea and malaise are the earliest findings of hyponatremia, and may be seen when the plasma sodium concentration falls below 125 to 130 meq/L. This may be followed by headache, lethargy, and obtundation Muscle weakness due to hypokalemia or hyperkalemia Polyuria and polydipsia due to hyperglycemia or severe hypokalemia Lethargy, confusion, seizures, and coma due to hyponatremia, hypernatremia, or hyperglycemia
Slide 31-

Basic signs of hypovolemia Urine output, less than 30ml/hr Decreased BP, Increase pulse
Slide 32-

Physical exam for assessing volume physical exam in general is not sensitive or specific acute weight loss; however, obtaining an accurate weight over time may be difficult decreased skin turgor - if you pinch it it stays put dry skin, particularly axilla dry mucus membranes low arterial blood pressure (or relative to patient's usual BP) orthostatic hypotension can occur with significant hypovolemia; but it is also common in euvolemic elderly subjects. decreased intensity of both the Korotkoff sounds (when the blood pressure is being measured with a sphygmomanometer) and the radial pulse ("thready") due to peripheral vasoconstriction. decreased Jugular Venous Pressure The normal venous pressure is 1 to 8 cmH2O, thus, a low value alone may be normal and does not establish the diagnosis of hypovolemia.
Slide 33-

SIGNS & SYMPTOMS OF Fluid Volume Excess SOB & orthopnea Edema & weight gain Distended neck veins & tachycardia Increased blood pressure Crackles & wheezes pleural effusion
Slide 34-

For the EBM aficionados out there. A JAMA 1999 systematic review of physical diagnosis of hypovolemia in adults CONCLUSIONS: A large postural pulse change (> or =30 beats/min) or severe postural dizziness is required to clinically diagnose hypovolemia due to blood loss, although these findings are often absent after moderate amounts of blood loss. In patients with vomiting, diarrhea, or decreased oral intake, few findings have proven utility, and clinicians should measure serum electrolytes, serum blood urea nitrogen, and creatinine levels when diagnostic certainty is required.
Slide 35-

Which brings us to: Labnormalities seen with hypovolemia a variety of changes in urine and blood often accompany extracellular volume depletion. In addition to confirming the presence of volume depletion, these changes may provide important clues to the etiology.
Slide 36-

BUN/Cr BUN/Cr ratio normally around 10 Increase above 20 suggestive of “prerenal state” (rise in BUN without rise in Cr called “prerenal azotemia.”) This happens because with a low pressure head proximal to kidney, because urea (BUN) is resorbed somewhat, and creatinine is secreted somewhat as well
Slide 37-

Hgb/Hct Acute loss of EC fluid volume causes hemoconcentration (if not due to blood loss) Acute gain of fluid will cause hemodilution of about 1g of hemoglobin (this happens very often.)
Slide 38-

Plasma Na Decrease in Intravascular volume leads to greater avidity for Na (through aldosterone) AND water (through ADH), So overall, Plasma Na concentration tends to decrease from 140 when hypovolemia present.
Slide 39-

Urine Na Urine Na – goes down in prerenal states as body tries to hold onto water. Getting a FENa helps correct for urine concentration. Screwed up by lasix. Calculator on PDA or medcalc.com
Slide 40-

IV Modes of administration Peripheral IV PICC Central Line Intraosseous
Slide 41-

IV Problem:Extravasation / “Infiltrated” The most sensitive indicator of extravasated fluid or "infiltration" is to transilluminate the skin with a small penlight and look for the enhanced halo of light diffusion in the fluid filled area. Checking flow of infusion does not tell you where the fluid is going
Slide 42-

That’s it folks.