Download Free Medical ENT presentation PowerPoint Presentation

ENT presentation PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical presentation on Otolaryngology, which is titled "ENT presentation", to know how to assess and manage common ENT problems in primary care.

Tags : Otolaryngology, Eye, Ear, Nose, Throat Problems

Published on : Jul 10, 2021 | Views : 1296 | Downloads : 14

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

ENT presentation Barbara Adams and Mike Pointon
Slide 2-

Aims and objectives Know how to assess and manage common ENT problems in primary care Know about watchful waiting and use of delayed prescriptions Know how and when to refer to ENT secondary care for non-urgent referrals Know about ENT emergencies and how to refer
Slide 3-

Ear
Slide 4-

Acute otitis media (AOM) definitions AOM: Infection in middle ear, characterised by presence of middle ear effusion associated with acute onset of signs and symptoms of middle ear inflammation Recurrent AOM: ≥3 episodes in 6m or ≥4 in 1y with absence of middle ear disease between episodes Persistent AOM (treatment failure): symptoms persist after initial management (no antibiotics, delayed antibiotics or immediate antibiotic prescribing strategy) or symptoms worsening
Slide 5-

AOM: causes & complications Bacterial infection: most common- strep pneumoniae, h influenzae (only 10% due to type B and preventable by HIB vaccine), moraxella catarrhalis Viral infection: most common- respiratory syncytial virus and rhinovirus Complications: hearing loss; chronic perforation and otorrhoea, CSOM, cholesteatoma, intracranial complications
Slide 6-

AOM: diagnosis Presents with earache (!) In younger children-non specific symptoms, e.g rubbing ear, fever, irritability, crying, poor feeding, restlessness at night, cough, or rhinorrhoea Mastoiditis AOM AOM
Slide 7-

AOM Differential diagnoses Other URTI: may be mild redness of TM, self limiting Otitis media with effusion (OME)/ glue ear: fluid in middle ear without signs of acute inflammation of TM CSOM: persistent inflammation and TM perforation with exudate >2-6w. May lead to . . . . . . Acute mastoiditis (rare)- swelling, tenderness and redness over mastoid bone, pinna pushed forward Bullous myringitis (rare)- haemorrhagic bullae on TM caused by Mycoplasma pneumoniae (90% spontaneous resolution)
Slide 8-

Management of AOM: when to refer or admit? Advise a no antibiotic or delayed antibiotic strategy for most people with suspected AOM but: consider antibiotics in children < 3m, bilateral AOM systemically unwell high risk of complications e.g. immunosuppression, CF. For all antibiotic prescribing strategies: inform patient average duration of illness for untreated AOM is 4 days. Admit: According to “Feverish illness in Children” NICE Guidance Adults and children with suspected complications e.g. meningitis, mastoiditis, or facial paralysis Amoxicillin or Erythromycin
Slide 9-

Follow up of AOM Routine follow up not usually required Follow up if: symptoms worse or not settling within 4 days otorrhoea persists >2w perforation if hearing loss persists in absence of pain or fever, ie OME Recurrent AOM: Second line co-amoxiclav http://guidance.nice.org.uk/CG47 Feverish illness in children http://guidance.nice.org.uk/CG69 Respiratory Tract Infections
Slide 10-

Otitis media with effusion (OME) / Glue ear Definition: non-purulent collection of fluid in middle ear (must be > 2/52 after recent AOM to be classed as Glue ear) Causes: Eustachian tube dysfunction > 50% due to AOM especially in < 3 yrs Other: low grade bacterial/viral infections; gastric reflux; nasal allergies; adenoids or nasal polyps; CF; Down’s Pressure changes e.g. with flying or scuba diving (adults) Symptoms: hearing loss absence of earache or systemic upset can present with problems of speech/language development, behaviour or social interaction
Slide 11-

Otitis media with effusion
Slide 12-

Other causes of hearing loss (or perceived loss)- Foreign body in EAC perforated TM SNHL listening problems inc ADHD and learning difficulty Initial management of OME Ask about developmental delay or language difficulties Hearing test Drugs not recommended as OME usually self limiting but consider ICS if there is associated allergic rhinitis
Slide 13-

Early intervention with grommets gives no benefit for long-term hearing, language and behaviour and increases risk of TM abnormalities. Subgroup with hearing loss > 25DB may benefit from early grommet insertion.
Slide 14-

OME general advice: good prognosis, self-limiting and >90% get resolution within 6m; limited proven benefit from drugs OME in adults is unusual in adults and need referral to ENT (unilateral could mean nasopharyngeal ca) Grommets – general points: usually stop functioning after 10m approx 50% require reinsertion within 5y conductive deafness after extrusion improves slowly Complications are otorrhoea, may need specialist input. most activities unaffected, i.e. can fly and swim but avoid immersion; re hearing loss should face child when speaking Adenoidectomy: is usually second line treatment for OME but no UK national guideline; conflicting evidence. No evidence for Tonsillectomy in OME
Slide 15-

Chronic Suppurative Otitis Media (CSOM) Symptoms persistent painless otorrhoea >2w May be preceded by AOM, trauma and grommets Differentials OE, FB, wax Assessment Exclude intracranial involvement, facial paralysis or mastoiditis- needs admission otherwise routine referral
Slide 16-

Otitis externa (OE) Inflammation of EAC Localised OE: folliculitis that can progress to a furuncle Diffuse OE: more widespread inflammation e.g. swimmers ear OE defined as: acute if episode<3w; chronic if >3m Malignant OE: extends to mastoid and temporal bones resulting in osteitis. Typically in elderly diabetics. Suspect if pain seems disproportionate to clinical findings
Slide 17-

Localised OE Causes: usually infected hair root by staph aureus Symptoms: severe ear pain (compared to size of lesion); relief if furuncle bursts; hearing loss if EAC very swollen Signs: tiny red swelling in EAC (early); later has white or yellow pus-filled centre which can completely occlude EAC Management: analgesia; hot compress; antibiotic only if severe infection or high risk patient - flucloxacillin or erythromycin Refer: if needs I+D, no response to antibiotic or cellulitis spreading outside EAC
Slide 18-

Acute diffuse OE Causes: bacterial infection- pseudomonas or staph aureus seborrhoeic dermatitis fungal infection- usually candida contact dermatitis - meds (sudden onset) or hearing aids/earplugs (insidious onset) Symptoms: any combination of ear pain, itch, discharge and hearing loss Signs: EAC and/or external ear are red, swollen or eczematous serous/purulent discharge inflamed TM – may be difficult to visualise pain on moving ear or jaw Investigations: rarely useful but if treatment fails, send swab for bacterial and fungal culture
Slide 19-

Management: Use topical ear preparation for 7 days; 2% acetic acid for mild cases antibiotic plus steroid e.g. Locorten-Vioform Gentisone HC (NB not if perforation) If wax/debris obstructing EAC or extensive swelling or cellulitis Pope wick Dry mopping (children) Microsuction (ENT PCC) Advise re prevention of OE: keep ears clean and dry; treat underlying eczema/psoriasis Failure of topical meds: review diagnosis/compliance consider PO fluclox or erythromycin ?fungal (spores in EAC) Swab and refer
Slide 20-

Chronic OE Causes: Secondary fungal infection- due to prolonged use of topical antibacterials or steroids Seborrhoeic dermatitis; contact dermatitis Sometimes no cause can be found for OE Symptoms: mild discomfort; pain usually mild Signs: lack of ear wax; dry, hypertrophic skin leading to canal stenosis; pain on exam Assess risk /precipitating factors; severity of symptoms; signs of fungal infection- whitish cotton-like strands in EAC, black or white balls of aspergillus. Look for signs of dermatitis, evidence of allergy (ear plugs etc) or focus of fungal infection elsewhere, e.g. Skin, nails, vagina- can cause 2’ inflammation EAC Investigations: only take swab for C+S if treatment fails as interpretation can be difficult: sensitivities are determined for systemic use and much higher concentrations can be achieved by topical use; organisms may be contaminants, usually fungal overgrowth after using antibacterial drops due to suppressed normal bacterial flora
Slide 21-

Chronic OE Management: advise general measures as for acute diffuse OE Treatment depends on cause - often requires more than one strategy: if fungal infection- top antifungal, refer if poor response seborrhoeic dermatitis- antifungal and steroid combined If no cause evident- 7d course top steroid +/- acetic acid spray. If good response, may need to continue steroid but reduce potency/dose. If cannot be withdrawn after 2-3m, refer ENT. If poor response, try trial of top antifungal Refer ENT if contact sensitivity (re patch testing); if EAC occluded; if malignant OE suspected.
Slide 22-

Foreign Bodies Management depends on what it is: Batteries – immediate referral to ENT Inert FB – e.g. retained grommet, beads, foam - not so urgent Organic – e.g. food, insects. May cause infection therefore should be dealt with sooner. For insects – drown in olive oil first. Some FBs may resolve with syringing, but if not refer to PCC Do not attempt to remove under direct visualisation as more likely to cause harm
Slide 23-

Nose
Slide 24-

Anterior or Posterior – hx gives clues > 90% from Little’s Area Age gives clue – more likely posterior in Elderly Cause: Idiopathic, trauma (nose picking), dry mucosa, hypertension, coagulopathy, NSAID, Warfarin, tumour CAN BE FATAL!!! First Aid: Compression & Ice Epistaxis
Slide 25-

Avoid blowing their nose (1/52) Avoid hot drinks (1/52) Naseptin cream 1/52 Admit: If cannot control, elderly, warfarinised, low platelets, recurrent excessive bleeding PCC: If not settling with conservative rx AgNO3 cautery – can be done in GP Packing, Electrocautery, Surgery (SPA ligation, ECA ligation, embolisation)
Slide 26-

ppt slide no 26 content not found
Slide 27-

Cautery: What you need: A good lightsource Nasal speculum (or large aural speculum) Lignocaine (with adrenalin) Cotton wool Cautery sticks
Slide 28-

Causative factors – allergic, viral, bacterial, fungal, autoimmune. Acute <12wks, Chronic >12wks, Recurrent (>4/yr) 15% population. 6 million lost working days / yr in the UK Presents as “My cold won’t go away” – persistant symptoms of URTI, without improvement after 10-14 days or worsening after 5 days Major: Nasal congestion/obstruction Purulent discharge Loss of smell Facial pain / ear pain or fullness Rhinosinusitis
Slide 29-

Minor: Tenderness over sinus area Fever Headache Halitosis Fatigue / Lethargy Post nasal drip What to exclude on examination: Periorbital swelling, extraocular muscle dysfunction, decreased VA or proptosis Foreign bodies Concomitant otitis media (in children) CNS complications Polypoid changes or deviated septum What to expect on examination: Erythema / swelling of nasal mucosa Mucopurulent secretions Tenderness over sinuses
Slide 30-

Differentials Allergic rhinitis (seasonal or perennial) Usually just nasal symptoms and usually persistent Nasal FB – unilateral blockage or discharge Sinonasal tumour – chronic, unilateral blockage, discharge (bloody) Other causes of facial pain Tension Headache TMJ dysfunction or bruxism Neuropathic Dental pain (hot/cold drinks, chewing) Investigations Xrays / Bloods / Swabs = not required, only indicated if > 12 wks and failure to respond to Rx – will probably refer at that stage (rigid endoscopy / coronal CT / allergy testing)
Slide 31-

Consider emergency admission to hospital if symptoms are accompanied by: Systemic illness Swelling or cellulitis in face Signs of CNS involvement Orbital involvement Consider urgent ENT referral if: Persistant unilateral symptoms such as (suspecting sinonasal tumour): Bloodstained discharge Non-tender facial pain Facial swelling Unilateral polyps Consider routine referral to ENT if: More than 3-4 episodes per year lasting > 10 days with no symptoms between episodes
Slide 32-

Management of acute rhinosinusitis (guidelines on map of medicine) Viral is 200 times more common than bacterial Viral URTI usually precedes bacterial Bacterial usually has more severe and prolonged symptoms Strep pneumoniae, H. influenzae, Moraxella Catarrhalis First line : Amoxicillin Doxycycline, erythromycin, clarithromycin (pen allergic) Second Line: Co-amoxiclav Azithromycin (pen allergic)
Slide 33-

ppt slide no 33 content not found
Slide 34-

Management of chronic rhinosinusitis (referral toolkit)
Slide 35-

Commonest in children aged 1-4 Rare in adults Potential risk to airway Suspect if persistant unilateral symptoms of blockage or foul smelling discharge Unless very easy to get at, and very compliant child, best not attempted in GP (sometimes only get one shot!) Nasal Foreign Bodies
Slide 36-

Best viewed from above – looking at deviation of nasal bones – difficult if swollen Exclude septal haematoma Requires immediate drainage to prevent abscess or permanent saddle nose deformity Otherwise refer to PCC for manipulation 7-10 days post injury. For old injuries routine ENT referral Nasal Fracture
Slide 37-

Nasal blockage will almost always be accompanied by snoring Have OSA in the back of your mind Defined as the presence of at least five obstructive events per hour during sleep Features Impaired alertness Cognitive impairment Excessive sleepiness (Epworth scale) Morning headaches Choking or SOB feeling at night Nocturia Unrefreshing sleep Sleep quality of partners affected (“does he stop breathing at night?”) Refer to Respiratory in the first instance Consider OSA
Slide 38-

Throat
Slide 39-

Sore throat: causes Common infections: rhinovirus; coronovirus, parainfluenza virus; common cold (25% sore throats) GABHS causes 15-30% sore throats in children and 10% in adults Herpes simplex virus type 1 (more rarely type 2) = 2% Epstein Barr virus: infectious mononucleosis (glandular fever)- <1%. Suspect IM if sore throat persists >2w - do FBC and IM screen. Non-infectious causes Physical irritation Hayfever Stevens Johnson syndrome Kawasaki disease Oral mucositis 2’ chemo /radiotherapy
Slide 40-

Sore throat: complications Complications of streptococcal pharyngitis are rare: Suppurative complications: OM acute sinusitis peritonsilar cellulitis / peritonsillar abscess (quinsy) Pharyngeal abscess Retropharyngeal abscess, more common in children Non suppurative complications are rare: rheumatic fever post-streptococcal glomerulonephritis R sided quinsy showing displacement of uvula to L
Slide 41-

Sore throat: when to refer Admit if stridor or respiratory difficulty Trismus, drooling, dysphagia. Dehydration /unable to take fluids Severe suppurative complications, ie if abnormal throat swelling/suspected abscess Systemically unwell and at risk of immunosuppression Suspect Kawasaki disease Profoundly unwell and cause unknown
Slide 42-

Sore throat: management in primary care Reassure sore throat usually self limiting and symptoms resolve within 3d in 40% cases, 1w in 85% (even if due to streptococcal infection) Advise see healthcare professional if symptoms do not improve, and urgently if breathing difficulties, stridor, drooling, muffled voice, severe pain, dysphagia or unable to take fluids or systemically ill Symptoms of infectious mononucleosis usually resolve within 1-2w, mild cases within days. But lethargy continues for some time and rarely may continue for months or years. Advise re contact sport. Advise regular paracetamol, ibuprofen, fluids ++ but avoid hot drinks; saline mouthwashes; discuss role of antibiotics Consider delayed prescription or immediate antibiotics – use Centor scoring - Antibiotic regime: Prescribe phenoxymethylpenicillin for 10d; or erythromycin or clarithromycin for 5d. Avoid amoxicillin (EBV)
Slide 43-

Indications for tonsillectomy for recurrent acute sore throat Sore throats are due to acute tonsillitis Episodes of sore throat are disabling and prevent normal functioning Seven or more well documented, clinically significant, adequately treated sore throats in the preceding year or Five or more such episodes in each of the preceding two years or Three or more such episodes in each of the preceding three years SIGN 2010, Management of sore throat and indications for tonsillectomy http://www.sign.ac.uk/pdf/qrg117.pdf
Slide 44-

Vertigo Vertigo: ‘is a symptom and refers to a perception of spinning or rotation of the person or their surroundings in the absence of physical movement’ Peripheral vertigo = labyrinthine cause Benign paroxysmal positional vertigo (BPPV) Vestibular neuronitis: Meniere’s disease: Central vertigo = cerebellar cause Common Migraine Uncommon stroke and TIA cerebellar tumour acoustic neuroma MS
Slide 45-

Assessment of vertigo Most balance problems that present in primary care are not rotatory vertigo, but unsteadiness. A full time GP is likely to see 10-20 people with vertigo in 1y To determine vertigo rather than dizziness, ask: “do you feel light-headed or do you see the world spin around you as if you had just got off a roundabout” about timing, duration, onset, frequency and severity of symptoms aggravating factors, e.g. head movement effect on daily activities associated symptoms: hearing loss, tinnitus (unilateral/bilateral), headache, diplopia, dysarthria /dysphagia, ataxia, nausea, vomiting
Slide 46-

Assessment of vertigo: medical history Recent URTI or ear infection suggests vestibular neuronitis or labyrinthitis Migraine: inc likelihood of migrainous vertigo Head trauma/ recent labyrinthitis: BPPV Trauma to ear: perilymph fistula Anxiety or depression can worsen symptoms or cause feelings of lightheadedness (e.g. from hyperventilation) Acute alcohol intoxication can cause vertigo Examination ENT – incl. Weber and Rinnes tests Full Neuro incl cerebellar testing + gait. Particularly looking for nystagmus
Slide 47-

Assessment of vertigo: specific tests Romberg’s test: identifies peripheral or central cause of vertigo (but not sensitive for differentiating between them) Ask patient to stand up straight, feet together, arms outstretched with eyes closed. If patient unable to keep balance- the test is positive (usually fall to side of lesion) A positive test suggests problem with proprioception or vestibular function. Hallpike manoeuvre: to confirm diagnosis of BPPV
Slide 48-

Hallpike manoeuvre - demonstration Be cautious with patients with neck or back pathology or carotid stenosis as manouvre involves turning and extending neck http://northerndoctor.com/2010/09/27/dizziness-dix-hallpike-and-the-epley-manoeuvre/ Ask patient to: report any vertigo during test keep eyes open and stare at examiner’s nose sit upright on couch, head turned 45’ to one side lie them down rapidly until head extended 30’ over end of bed, one ear downward If neck problems- can be done without neck extension observe eyes closely for 30 sec for nystagmus- note type and direction support head in position and sit up Repeat with other side test is positive for BPPV if vertigo and nystagmus (torsional and beating towards ground) are present and nystagmus shows latency, fatigue and adaptation
Slide 49-

Features of central causes of vertigo severe or prolonged new onset headache focal neurological deficits central type nystagmus (vertical) excess nausea and vomiting prolonged severe imbalance (inability to stand up even with eyes open)
Slide 50-

Features of peripheral causes of vertigo BPPV: vertigo induced by moving head position episodes last for seconds Vestibular neuronitis and labyrinthitis: vertigo persists for days and improves with time no hearing loss or tinnitus with vestibular neuronitis in labyrinthitis, sudden hearing loss with vertigo and tinnitus may be present Meniere’s disease: ages 20-50y men> women vertigo, not provoked by position change episodes last 30 min to several hours symptoms of tinnitus, hearing loss and fullness in ear may be clusters of attacks and long remissions
Slide 51-

Medication used in vertigo prochlorperazine cyclizine cinnarizine promethazine
Slide 52-

Tinnitus Unwanted perception of sound within head, in absence of sound from external environment Can be described as ringing, hissing, buzzing, roaring or humming. Classified as- Subjective tinnitus: sound only heard by patient; assoc with abnormalities of auditory system Objective tinnitus: sound heard by patient and examiner; caused by physical abnormality that produces sound near or within ear
Slide 53-

Disorders associated with subjective tinnitus Two thirds people with tinnitus have disorder causing hearing loss; one third have idiopathic tinnitus Most commonly assoc with disorders causing sensorineural hearing loss (SNHL): age related noise induced Meniere’s disease Less commonly assoc with disorders causing conductive hearing loss: impacted wax otosclerosis (rare)
Slide 54-

Uncommonly, subjective tinnitus is associated with: Ototoxic drugs Cytotoxic drugs (e.g. Cisplatin, methotrexate) Aminoglycosides (gentamicin) macrolides, quinine, aspirin, NSAIDs and loop diuretics Ear infections: (OM, OME, CSOM) Neurological disorders: acoustic neuroma; schwannoma, MS Metabolic disorders: Hypothyroidism; diabetes Psychological disorders: anxiety and depression Trauma
Slide 55-

Disorders associated with objective tinnitus Objective tinnitus is very rare Due to: Vascular disorders: AVMs; vascular tumours; High output states: anaemia; thyrotoxicosis; Paget’s disease
Slide 56-

Management of tinnitus in primary care Assess underlying cause Refer to ENT: All patients with objective tinnitus Patients with subjective tinnitus, following hearing test, who have associated SNHL Tinnitus associated with conductive hearing loss when treatable causes not identified or managed in primary care Tinnitus secondary to head or neck injury Tinnitus of uncertain cause Tinnitus that is causing distress despite primary care management
Slide 57-

Foreign Bodies Feeling of food (most commonly) stuck in throat / oesophagus If complete dysphagia of acute onset, then very high chance of a FB obstruction If delayed onset of FB sensation after eating, and mild symptoms, could simply be abrasion, symptoms will go in 48 hrs. Refer if not resolved Oesophageal food bolus: coke or pineapple juice, buscopan (IM) or GTN (SL) can help
Slide 58-

Other
Slide 59-

Lower motor neurone (involving forehead) Motor supply to the scalp, facial muscles & stapedius Taste to anterior 2/3 of the tongue VII nerve palsy
Slide 60-

Possible causes: Traumatic facial lacerations, blunt trauma ( BOS fracture), newborn paralysis Neoplastic parotid tumors, tumors of the external canal and middle ear, metastatic lesions, SCC, cholesteatoma, acoustic neuroma Infectious herpes zoster oticus (Ramsey-Hunt syndrome), AOM, CSOM, malignant otitis externa Idiopathic Bell's palsy although traditionally defined as idiopathic it is thought to be associated with herpes simplex virus type 1
Slide 61-

Characteristics of a peripheral facial paralysis include: Motor unable to wrinkle forehead unable to raise eyebrow unable to wrinkle nasolabial fold unable to purse lips or show teeth inability to completely close eye (classified using House-Brackmann scale) Decreased taste sensation Hyperacusis Reduction of lacrimation
Slide 62-

Need full head & neck examination If Ramsey-Hunt will give aciclovir All will get steroids (40mg prednisolone daily) Eye taping at night and lacrilube if cannot close eye Referral to PCC Will get hearing test on the day and subsequent follow up +/- Ophthalmology referral Prognosis depends on cause
Slide 63-

Sialolithiasis (calculi) Sialadenitis (inflammation) Acute Chronic Recurrent Tumours Other Salivary Gland Problems
Slide 64-

Examination Inspect the enlarged gland and all others Tender – Sialadenitis / Sialolithiasis Non-Tender – Tumour More than one gland affected – autoimmune or viral (e.g. Mumps) Overlying inflammation might point towards infection Test facial nerve Inspect the oral cavity (bimanual) May be able to palpate a stone May be able to express pus from the duct
Slide 65-

80-95% in SMG, 5-20% in Parotid Intermittent pain and swelling at meal times. Acidic or spicy foods cause worse symptoms Swelling appears before, and persists after the pain Most common in 3rd – 6th decades Very rarely cause complete salivary obstruction Sialolithiasis
Slide 66-

Palpation of SMG SMG duct (Wharton’s) openings Stone inside duct opening
Slide 67-

Opening of Parotid Duct (Stensen’s) Adjacent to maxillary 2nd molar
Slide 68-

Management Sour foods (sialogogues) to stimulate saliva flow Massaging the affected gland to promote saliva flow Artificial saliva products and/or frequent small drinks Antibiotics may be required for episodes of acute inflammation (see Sialadenitis) Refer if not settling
Slide 69-

Most commonly affects the Parotid (Parotitis) Elderly, dehydrated, debilitated Pain & fever Tender swelling with redness, may be purulent discharge from the duct Sialadenitis
Slide 70-

Management Rehydration Staph aureus is most common organism Flucloxacillin Co-amoxiclav Refer for admission if: Fails to improve after 5/7 ABx Facial nerve involvement Requiring IV fluids
Slide 71-

Prophylaxis Adequate fluid intake Avoidance of anticholinergics Good oral hygiene (gargles etc) Stimulation of salivation e.g. gum chewing Chronic Usually from partial duct obstruction Refer Recurrent Consider swabbing any duct discharge Refer
Slide 72-

Usually more insidious onset Usually painless Going to be referring under 2ww rules for neck lump Tumours
Slide 73-

Autoimmune – Sjogren’s Metabolic – Myxoedema, DM, Cushing’s, Bulimia, Alcoholism, Cirrhosis, Gout Drug induced – OCP, Coproxamol Viral – Mumps Other
Slide 74-

Referrals (only 5 slides to go . . . . . . )
Slide 75-

Same day – SHO Primary Care Clinic - SHO 2 week wait – faxed Routine referrals – Voice/Balance/General/Thyroid/Oncology – written/C&B Audiology – written/C&B Microsuction – written/C&B ENT SHO through switchboard or bleep 585 Ward 15 (Adults) or Ward 17/18 (Children) Referrals
Slide 76-

Located in Head & Neck outpatients YDH Accessed through SHO AM & PM Mon, Tues, Thu, Fri Usually will get appt within a week, sooner if clinical need. SHO led with support from Staff Grades / SpR Have access to audiometry on the day Primary Care Clinic
Slide 77-

Otitis Externa Nasal Fracture Epistaxis VII n palsy Recent parotid swelling (stones/infection) Sudden SNHL Foreign bodies Submandibular swellings usually go via max facs to exclude dental abscess
Slide 78-

NICE Guidance CG27 June 2005 Refer urgently patients with: an unexplained lump in the neck, of recent onset, or a previously undiagnosed lump that has changed over a period of 3 to 6 weeks an unexplained persistent swelling in the parotid or submandibular gland an unexplained persistent sore or painful throat unilateral unexplained pain in the head and neck area for more than 4 weeks, associated with otalgia (ear ache) but a normal otoscopy unexplained ulceration of the oral mucosa or mass persisting for more than 3 weeks unexplained red and white patches (including suspected lichen planus) of the oral mucosa that are painful or swollen or bleeding For patients with persistent symptoms or signs related to the oral cavity in whom a definitive diagnosis of a benign lesion cannot be made, refer or follow up until the symptoms and signs disappear. If the symptoms and signs have not disappeared after 6 weeks, make an urgent referral. Two-Week-Rule
Slide 79-

Hoarseness > 3/52  CXR  ENT if NAD Refer urgently patients with a thyroid swelling associated with any of the following: a solitary nodule increasing in size a history of neck irradiation a family history of an endocrine tumour unexplained hoarseness or voice changes cervical lymphadenopathy very young (pre-pubertal) patient patient aged 65 years and older Do not delay referral with Ix (e.g. TFTs / USS) Request thyroid function tests in patients with a thyroid swelling without stridor or any of the features listed above. Refer patients with hyper-/hypo-thyroidism and an associated goitre, non-urgently, to an endocrinologist. Patients with goitre and normal thyroid function tests without any of the features listed above should be referred non-urgently http://guidance.nice.org.uk/CG27