Download Free Medical Classification and management of wound principle of wound healing haemorrhage and bleeding control PowerPoint Presentation

Classification and management of wound principle of wound healing haemorrhage and bleeding control PowerPoint Presentation

DOWNLOAD MEDICAL POWERPOINT PRESENTATION

About This Presentation

Description : Check out this medical PowerPoint presentation titled "Classification and management of wound, principle of wound healing, haemorrhage and bleeding control" by Györgyi Szabó.

This medical PowerPoint presentation talks about classification and management of wound. It also lists principle of wound healing, haemorrhage and bleeding control.

Tags : wound healing, haemorrhage control, bleeding control, classification of wound, management of wound, Emergency Medicine, Emergency Medicine PowerPOint presentation

Published on : Apr 12, 2023 | Views : 456 | Downloads : 4

Shareable Url :

Embed Code :

Comment

Transcript

Slide 1-

Györgyi Szabó Assistant Professor Department of Surgical research and Techniques Classification and management of wound, principle of wound healing, haemorrhage and bleeding control Basic Surgical Techniques, Faculty of Medicine, 3rd year 2021/13 Academic Year, Second Semester 1
Slide 2-

WOUND 2
Slide 3-

What is a wound? It is a circumscribed injury which is caused by an external force and it can involve any tissue or organ. surgical, traumatic It can be mild, severe, or even lethal. Simple wound Compound wound Acute Chronic 3
Slide 4-

Parts of the wound 4
Slide 5-

The ABCDE in the injured assessment The mnemonic ABCDE is used to remember the order of assessment with the purpose to treat first that kills first. A: Airway and C-spine stabilization B: Breathing C: Circulation D: Disability E: Environment and Exposure 5
Slide 6-

Wound management - anamnesis When and where was the wound occured? Alcohol and drug consumption What did caused the wound? The circumstances of the injury Other diseases eg. diabetes mellitus, tumour, atherosclesosis, allergy The state of patient’s vaccination against Tetanus Prevention of rabies The applied first-aid 6
Slide 7-

Classification of the accidental wounds1. Based on the origine I. Mechanical: 1. Abraded wound (vulnus abrasum) 2. Puncured wound (v. punctum) 3. Incised wound (v. scissum) 4. Cut wound (v. caesum) 5. Crush wound (v. contusum) 6. Torn wound (v. lacerum) 7. Bite wound (v. morsum) 8. Shot wound (v. sclopetarium) II. Chemical: 1. Acid 2. Base III. Wounds caused by radiation IV. Wounds caused by thermal forces: 1. Burning 2. Freezing V. Special 7
Slide 8-

1.) Abraded wound (v. abrasum) 2.) Punctured wound (v. punctum) Superficial part of the epidermal layer Good wound healing Sharp-pointed object Seems negligible BUT Anaerobic infection Injury of big vessels and nerves Mechanical wounds 8
Slide 9-

3.) Incised wound (v. scissum) 4.) Cut wound (v. caesum) Sharp object Best healing Sharp object + blunt additional force Edges - uneven Mechanical wounds 9
Slide 10-

5.) Crush wound (v. contusum) 6.) Torn wound (v. lacerum) Blunt force Pressure injury Edges – uneven and torn Bleeding Great tearing or pulling Incomplete amputation Mechanical wounds 10 (v. lacerocontusum)
Slide 11-

7.) Shot wound (v. scolperatium) Close - burn injury Foreign materials Mechanical wound 11 unijured tissue necrobiotic zone necrotic zone foreign bodies aperture slot tunel output
Slide 12-

8.) Bite wound (v. morsum) Ragged wound Crushed tissue Torn Infection Bone fracture Prevention of rabies Tetanus profilaxis Mechanical wounds 12
Slide 13-

Distal Proximal The wound healing is good The direction of the flap 13
Slide 14-

1.) Acid 2.) Base in small concentration – irritate in large concentration – coagulation necrosis colliquative necrosis Chemical wounds 14
Slide 15-

Symptoms and severity depend on: Amount of radiation Length of exposure Body part that was exposed Symptoms may occur immediately, after a few days, or even as long as months. What part of the body is most sensitive during radiation sickness? bone marrow gastrointestinal tract Wounds caused by radiation 15
Slide 16-

1.) Burning 2.) Freezing a – normal skin 1 - 1st degree – superficial injury (epidermis) 2 – 2nd degree –partial or deep partial thickness (epidermis+superficial or deep dermis) 3 – 3rd degree – full thickness (epidermis + entire dermis) 4 – 4th degree – (skin + subcutaneous tissue + muscle and bone) Treatment: Cooling – cold water and clean covering Wounds caused by thermal forces 16 Metabolic change! - toxemia mild, moderate, severe (redness, bullas, necrosis) rewarm – not only the frozen area but the whole body
Slide 17-

Exotic, poisonous animals Toxins, venom - toxicologist Skin necrosis Special wounds 17
Slide 18-

Classification of the wounds2. According to the bacterial contamination Clean wound Clean-contaminated wound Contaminated wound Heavily contaminated wound 18
Slide 19-

Superficial Partial thickness Full thickness Deep wound Classification of the wounds2. Depending on the depth of injury + bone, opened cavities, organs…etc. 19 source: http://www.funscrape.com/Search/1/skin+layers.html
Slide 20-

Wound management - history Ancient Egypt – lint (fibrous base-wound site closure), animal grease (barrier) and honey (antibiotic) „closing the wound preserved the soul” Greeks – acute wound= „fresh” wound; chronic wound = „non-healing” wound maintaining wound-site moisture Ambroise Paré – hot oil  oil of roses and turpentine, ligature of arteries instead of cauterization Lister pretreated surgical gauze – Robert Wood Johnson 1870s; gauze and wound dressings treated with iodide 20
Slide 21-

Applied wound management - colour continuum black black-yellow yellow yellow-red red red-pink pink 21 source: Applied wound management supplement – www.wounds-uk.com
Slide 22-

Applied wound managementinfection continuum contamination colonisation infection sterility critical colonisation 22 the quantity and diversity of microbes source: Applied wound management supplement – www.wounds-uk.com
Slide 23-

Applied wound managementexudate continuum Viscosity 23 source: Applied wound management supplement – www.wounds-uk.com
Slide 24-

The wound managemanet Temporary wound management (first aid) clean, hemostasis, covering Final primary wound management clean, anaesthesis, excision, sutures ALWAYS: thoracic cavity, abdominal wall or dura mater injury NEVER: war injury, inflammation, contamination, foreign body, special jobs, bite, shot, deep punctured wound Primary delayed suture (3-8 days) clean, wash – saline, cover excision of wound edges, sutures 24
Slide 25-

The wound managemanet 25 Early secondary wound closure (2 weeks) after inflammation, necrosis – proliferation anesthesia, refresh wound edges, suturing and draining Late secondary wound closure (4-6 weeks) anesthesis, scar excision, suturing, draining greater defect – plastic surgery
Slide 26-

The surgical wound Surgical incision Stretch and fix Handling the scalpel Langer lines Skin edges Vessels and nerves Hemostasis Langer lines Handling the scalpel 26 source: http://www.med-ars.it/galleries/langer.htm
Slide 27-

Tissue unifying and dressing the wound Skin: Stiches Clips Steri-Strips Tissue glues Fascia and subcutaneous layers: Interrupted stiches Fat – fat necrosis! Dressing: sterile, moist, antibiotic-containing, non-allergic, non-adhesive 27
Slide 28-

The wound healing Hemostasis-inflammation Granulation-proliferation Remodelling 28 http://www.worldwidewounds.com/2004/august/Enoch/images/enochfig1.jpg
Slide 29-

The main steps of the wound healing 1. Hemostasis-inflammation vasoconstriction fibrin clot formation proinflammatory citokines and growth factors releasing vasodilatation infiltration PMNs, macrophages cytokines releasing → angiogensis → fibroblast activation → B- and T-cells activation → keratinocytes activation → wound contraction 29 2. Granulation-proliferation fibroblast migration collagen deposition angiogensis granulation tissue formation epithelisation contraction 3. Remodelling regression of many capillaries physical contraction – myofibroblasts collagen degeneration and synthetisation new epithelium tensile strength – max. 80%
Slide 30-

Types of wound healing Healing by primary intention Healing by secondary intention Healing by tertiary intention 30 source: http://quizlet.com/13665246/chapter-3-tissue-renewal-regeneration-and-repair-flash-cards/
Slide 31-

Factors affecting wound healing Local Ischemia Infection Foreign body Edema, elevated tissue pressure Systemic Age and gender Sex hormones Stress Ischemia Diseases Obesity Medication Alcoholism and smoking Immunocompromised conditions Nutrition Hyperbaric oxygen treatment 31
Slide 32-

Complications of wound healingI. Early complications Seroma Hematoma Wound disruptin Superficial wound infection Deep wound infection Mixed wound infection 32
Slide 33-

1.) Seroma 2.) Hematoma Filled with serous fluid, lymph or blood Fluctuation, swelling, redness, tenderness, subfebrility TREATMENT: Sterile punture and compression Suction drain Early complications of wound healing 33 Bleeding, short drainage time, anticoagulant Risk of infection Swelling, fluctuation, pain, redness TREATMENT Sterile puncture Surgical exploration
Slide 34-

3.) Wound disruption A. partial – dehiscenece B. complete - disruption Surgical error Increased intraabdominal pressure Wound infection Hypoproteinaemia TREATMENT: U-shaped sutures Early complications of wound healing 34
Slide 35-

1.) Diffuse 2.) Localized Located below the skin TREATMENT Resting position Antibiotic Dermatological consultation Anywhere TREATMENT Surgical exploration Drainage X-ray examination Early complications of wound healingSuperficial wound infection 35 e.g. erysipelas e.g. abscess
Slide 36-

1.) Diffuse 2.) Localized TREATMENT Surgical exploration Open therapy H2O2 and antibiotics e.g. anaerobic necrosis Inside the tissues or body cavities TREATMENT surgical exploration drainage Early complications of wound healingDeep wound infection 36
Slide 37-

Mixed wound infection e.g. gangrene necrotic tissues putrid and anaerobic infection a severe clinical picture TREATMENT aggresive surgical debridement effective and specified (antibiotic) therapy 37 Complications of wound healingI. Early complications
Slide 38-

Complications of wound healingII. Late complications Hyperthrophic scar Keloid formation Necrosis Inflammatory infiltration Abscesses Foreign body containing abscesses 38
Slide 39-

Hypertrophic scar Keloid Develop in areas of thick chorium Non-hyalinic collagen fibres and fibroblasts Confine to the incision line TREATMENT Regress spontaneously (1-2 yrs) Late complications 39 Mostly African and Asian population Well-defined edge Emerging, tough structure Overproliferation of collagen fibers in the subcutaneous tissue Subjective complains TREATMENT Postoperative radiation Corticosteroid + local anaesthetic injection
Slide 40-

BLEEDING AND HEMOSTASIS 40
Slide 41-

Anatomical Diffuse Arterial – bright red, pulsate Venous – dark red, continuous Capillary – can become serious Parenchymal Bleeding 41
Slide 42-

Bleeding Severity of bleeding – the volume of the lost blood and time 42 source: http://lifeinthefastlane.com/2012/03/trauma-tribulation-025/
Slide 43-

The direction of hemorrage External Internal In a luminar organ (hematuria, hemoptoe, melena) In body cavities (intracranial, hemothorax, hemascos, hemopericardium, hemarthros) Among the tissues (hematoma, suffusion) 43
Slide 44-

Bleeding Preoperative hemorrhage Prehospital care! – maintenance of the airways, ventillation and circulation bandages, direct pressure, turniquets Intraoperative hemorrhage anatomical and/or diffuse depending on the surgeon, the surgery, position, the size of the vessel, pressure in the vessel ANESTHESIA! Postoperative bleeding ineffective local hemostasis, undetected hemostatic defect, consumptive coagulopathy or fibrinolysis 44
Slide 45-

Local General Hematoma, suffusion, ecchymosis Compression in the pleural cavity, in pericardium, in the skull Functional disturbancies – e.g. hyperperistalsis Pale skin, cyanosis, decreased BP. and tachycardia, difficulty in breeding, sweeting, decreased body temperature, unconsciousness, cardiac and laboratory standstill, laboratory disorders, signs of shock Signs of the bleeding 45
Slide 46-

Surgical hemostasis Aim – to prevent the flow of blood from the incised or transected vessels Mechanical methods Thermal methods Chemical and biological methods 46
Slide 47-

Surgical hemostasisMechanical methods Digital pressure – direct pressure, e.g. Pringle maneuver Tourniquet Ligation Suturing Preventive hemostasis Clips Bone wax other 47
Slide 48-

Thermal methods Low temperature Hypothermia – eg. stomach bleeding Cryosurgery dehidratation and denaturation of fatty tissue decreases the cell metabolism vasoconstriction 48
Slide 49-

Thermal methods High temperature Electrosurgery – electrocauterization Monopolar diathermy Bipolar diathermy Laser surgery coagulation and vaporization for fine tissues 49
Slide 50-

Thermal methods High temperature Electrocoagulation Electrofulguration (A) Electrodessication Electrosection 50
Slide 51-

Hemostasis with chemical and biological methods vasoconstriction coagulation hygroscopic effect Absorbable collagen Absorbable gelatin Microfibrillar collagen Oxidized celluloze Oxytocin Epinephrine Thrombin Hemcon QuikClot 51
Slide 52-

Hemostasis with chemical and biological methods HemCon 52